Clomiphene Citrate, commonly referred to as Clomid is a Selective Estrogen Receptor Modulator (SERM) used by many performance athletes for the purpose of Post Cycle Therapy (PCT). Originally developed for ovarian stimulation, as is common another use was found and as it stands today PCT planning and implementation may indeed be its most common purpose. Through the effects of Clomid there can be many benefits to the performance enhancer as we will shortly see but most importantly of all revolves around testosterone production. This effect is not only useful in-terms of re-stimulating production after a cycle but the effects of Clomid can also be very beneficial to the individual who simply suffers from low testosterone in a therapeutic sense.
As a SERM by its design Clomid prevents estrogen from binding to the receptors by binding in its place thereby preventing estrogen from performing in an undesirable manner. It is commonly believed that the effects of Clomid include reducing the total amount of estrogen you have in your body; this is not true. Clomid does not reduce nor prohibit estrogen from existing; it simply binds in its place. To have a reducing and prohibiting effect one would need to supplement with other medications such as Aromatase Inhibitors. By its nature the use of Clomid in conjunction with aromatizing anabolic androgenic steroids could aid in preventing the common side-effect known as Gynecomastia as this occurrence is caused by estrogen binding to the receptors in the pectoral region. However, while possible those who are sensitive will not find it strong enough, for the binding effects of Clomid while strong are often not strong enough to prevent such measure.
Of the various effects of Clomid most understand the prominent one is the stimulation of natural testosterone production. Clomiphene Citrate actively stimulates the gonadotropins by way of stimulating the release of Luteinizing and Follicle Stimulating Hormones (LH & FSH). Both LH and FSH are imperative to natural testosterone production with LH being primary, for without no testosterone can be produced. As the effects of Clomid stimulate the release of both LH and FSH we have two separate advantages both revolving around testosterone. For the performance athlete who is coming off cycle this allows him to bring his natural testosterone production back online sooner and with more efficiency, thereby allowing for more gains to be kept as well as maintenance of overall health. Further, for the individual suffering from low testosterone by way of natural causes, through supplemental use of this SERM he may indeed be able to provide his body with the testosterone he has been lacking through this stimulating effect. It is important to note, while Clomid serves its purpose very well during PCT, for the HRT patient the effects of Clomid are often not strong enough and direct exogenous testosterone therapy must be applied.
Nolvadex is another very popular SERM and we can aptly say the effects of Clomid are by and large identical to those of Nolvadex. Both SERMs actively bind to the receptors preventing estrogen from binding and both greatly stimulate the release of LH and FSH thereby promoting natural testosterone production. Both Nolvadex and Clomid can be used on-cycle for Gynecomastia prevention and both can be used effectively for PCT purposes as well as for testosterone replacement therapy. There is a good chance youve been told that Nolvadex or Nolva as it is often called is stronger than Clomid and even though the effects of Clomid and Nolva are identical in many ways this assumption is true; however, it doesnt have to be. The bottom line is simple; on a milligram for milligram basis Nolvadex is stronger than Clomid; when looking at these two SERMs, while simplistic you could easily look at Nolva as extra strength. When we adjust the doses to match in-terms of potency there really isnt much difference; so little of a difference the effects of Clomid vs. Nolvadex would be immeasurable by most.
